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> The mechanism behind type 2 diabetes is well known and it's all about eating too much carbs.

No it's not.

From https://www.ncl.ac.uk/magres/research/diabetes/reversal/#bac...

"We now know that type 2 diabetes is caused by excess fat inside liver and pancreas. ... The Twin Cycle Hypothesis described how it might be possible to explain the cause of type 2 diabetes in a very simple way. ... It was clear from that time onwards that type 2 diabetes is caused by too much fat building up within the liver, then overspilling to the rest of the body - including the pancreas. This starts up a second vicious cycle inside the pancreas, with the fat actually switching off normal insulin production...One of the most important discoveries is that of the Personal Fat Threshold. Type 2 diabetes is not caused by ‘obesity’. Different people have different levels of tolerance of fat within liver and pancreas. But if you have type 2 diabetes, you have crossed your ‘personal fat threshold’.



The explanation for T2D from the PDF paper from your own link explicitly singles out carbs and explains why they are critical to causing the disease process you describe.

> During any one period of time, if more calories are ingested than metabolized then any fat excess is stored either subcutaneously, viscerally or in the liver. But any excess carbohydrate cannot be stored once the glycogen depots are full. If more glucose is ingested than can be oxidized for energy or stored as glycogen, it has to be turned into fat by the process of de novo lipogenesis. This process only happens in the liver in humans, and triglyceride synthesized in situ is particularly likely to be stored in hepatocytes rather than exported for safe storage in subcutaneous adipose tissue.

> The newly synthesized fat has three possible fates: it can be oxidized for energy; exported as VLDL in the plasma to be delivered to other tissues or it can be stored in a rather full liver. As de novo lipogenesis is stimulated by insulin, those people who are relatively insulin resistant in muscle—and who therefore have a raised plasma insulin level—are especially likely to accumulate fat in the liver. This could explain the reason why muscle insulin resistance is the first detectable signal of risk for Type 2 diabetes.

https://www.ncl.ac.uk/media/wwwnclacuk/newcastlemagneticreso...




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