But what about the CJD controls that didn't receive transplants derived from cadaver tissue? Those didn't show the same rate of Alzheimers-like plaques, if I understood the report correctly.
The CJD controls had sporadic (sCJD), not variant CJD (vCJD) (whether or not these are distinct diseases is certainly an open question - I'm more trying to make the point that there are a lot of variables up in the air, and while it's a striking relationship there are other equally striking relationships one could draw).